Purine nucleoside phosphorylase

purine-nucleoside phosphorylase
purine-nucleoside phosphorylase. PDB 1rct.[1]
Identifiers
EC no.2.4.2.1
CAS no.9030-21-1
Databases
IntEnzIntEnz view
BRENDABRENDA entry
ExPASyNiceZyme view
KEGGKEGG entry
MetaCycmetabolic pathway
PRIAMprofile
PDB structuresRCSB PDB PDBe PDBsum
Gene OntologyAmiGO / QuickGO
Search
PMCarticles
PubMedarticles
NCBIproteins
PNP
Available structures
PDBOrtholog search: PDBe RCSB
Identifiers
AliasesPNP, NP, PRO1837, PUNP, purine nucleoside phosphorylase
External IDsOMIM: 164050; MGI: 97365; HomoloGene: 227; GeneCards: PNP; OMA:PNP - orthologs
EC number2.4.2.1
Orthologs
SpeciesHumanMouse
Entrez

4860

18950

Ensembl

ENSG00000198805

ENSMUSG00000115338

UniProt

P00491

P23492

RefSeq (mRNA)

NM_000270

NM_013632

RefSeq (protein)

NP_000261

NP_038660

Location (UCSC)Chr 14: 20.47 – 20.48 MbChr 14: 51.17 – 51.2 Mb
PubMed search[4][5]
Wikidata
View/Edit HumanView/Edit Mouse

Purine nucleoside phosphorylase, PNP, PNPase or inosine phosphorylase (EC 2.4.2.1) is an enzyme that in humans is encoded by the PNP gene.[6] It catalyzes the chemical reaction

Purine Nucleoside + Inorganic Phosphate (Pi) Purine Base + α-D-Ribose 1-Phosphate

The enzyme catalyzes reversible interconversion of purine nucleoside and phosphate into purine base and α-D-ribose 1-phosphate.

Nomenclature

This enzyme belongs to the family of glycosyltransferases, specifically the pentosyltransferases. The systematic name of this enzyme class is purine-nucleoside:phosphate ribosyltransferase.

Other names in common use include:

  • inosine phosphorylase
  • PNPase
  • PUNPI
  • PUNPII
  • inosine-guanosine phosphorylase
  • nucleotide phosphatase
  • purine deoxynucleoside phosphorylase
  • purine deoxyribonucleoside phosphorylase
  • purine nucleoside phosphorylase
  • purine ribonucleoside phosphorylas

This enzyme participates in 3 metabolic pathways: purine metabolism, pyrimidine metabolism, and nicotinate and nicotinamide metabolism.

Function

Purine nucleoside phosphorylase is an enzyme involved in purine metabolism. PNP metabolizes inosine into hypoxanthine and guanosine into guanine, in each case creating ribose-1-phosphate. In humans, adenosine is first metabolized to inosine via the enzyme adenosine deaminase.[7]

Nucleoside phosphorylase is an enzyme which cleaves a nucleoside by phosphorylating the ribose to produce a nucleobase and ribose-1-phosphate. It is one enzyme of the nucleotide salvage pathways. These pathways allow the cell to produce nucleotide monophosphates when the de novo synthesis pathway has been interrupted or is non-existent (as is the case in the brain). Often the de novo pathway is interrupted as a result of chemotherapy drugs such as methotrexate or aminopterin.

All salvage pathway enzymes require a high energy phosphate donor such as ATP or PRPP. For pyrimidine nucleosides:

Adenosine uses the enzyme adenosine kinase, which is a very important enzyme in the cell. Attempts are being made to develop an inhibitor for the enzyme for use in cancer chemotherapy.

Enzyme regulation

PNP protein may use the morpheein model of allosteric regulation.[8]

Clinical significance

Purine nucleoside phosphorylase, together with adenosine deaminase (ADA), serves a key role in purine catabolism. Mutations in ADA lead to an accumulation of dATP, which inhibits ribonucleotide reductase, leading to a deficiency in dCTP and dTTP, which, in turn, induces apoptosis in T-lymphocytes and B-lymphocytes, leading to severe combined immunodeficiency (SCID).[9]

PNP-deficient patients will have an immunodeficiency problem. It affects only T-cells; B-cells are unaffected by the deficiency.

See also

References

  1. ^ Canduri F, dos Santos DM, Silva RG, Mendes MA, Basso LA, Palma MS, et al. (January 2004). "Structures of human purine nucleoside phosphorylase complexed with inosine and ddI". Biochemical and Biophysical Research Communications. 313 (4): 907–914. Bibcode:2004BBRC..313..907C. doi:10.1016/j.bbrc.2003.11.179. PMID 14706628.
  2. ^ a b c GRCh38: Ensembl release 89: ENSG00000198805Ensembl, May 2017
  3. ^ a b c GRCm38: Ensembl release 89: ENSMUSG00000115338Ensembl, May 2017
  4. ^ "Human PubMed Reference:". National Center for Biotechnology Information, U.S. National Library of Medicine.
  5. ^ "Mouse PubMed Reference:". National Center for Biotechnology Information, U.S. National Library of Medicine.
  6. ^ "Entrez Gene: NP nucleoside phosphorylase".
  7. ^ Kaplan USMLE Biochemistry Review
  8. ^ Selwood T, Jaffe EK (March 2012). "Dynamic dissociating homo-oligomers and the control of protein function". Archives of Biochemistry and Biophysics. 519 (2): 131–143. doi:10.1016/j.abb.2011.11.020. PMC 3298769. PMID 22182754.
  9. ^ Akeson AL, Wiginton DA, States JC, Perme CM, Dusing MR, Hutton JJ (August 1987). "Mutations in the human adenosine deaminase gene that affect protein structure and RNA splicing". Proceedings of the National Academy of Sciences of the United States of America. 84 (16): 5947–5951. doi:10.1073/pnas.84.16.5947. PMC 298980. PMID 3475710.

Further reading